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This can directly verify the central retinal artery affected by L-NAME (i.e., vasoconstriction), with adequate time to produce/initiate the chain of damaging events, further lesion progress, and progressive NO-system failure from week 1 to week 2 to week 4 in L-NAME-treated rats [13]

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Genes that are optimal for the normal functioning of glutamate receptors and neuronal migration are found to be mutated or disrupted in ASD ( Fig