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Confirming the importance of oxidative stress in the mitochondrial DNA deletions, inhibition of monoamine oxidase B (MAO-B), an enzyme that metabolizes the monoamine neurotransmitters yielding H 2 O 2 as a byproduct, could avoid the oxidative damage to DNA (Alves et al., 2007)

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For management reasons, at 12:00 pm the family groups were reunited